IRIS publication 190495889
Host-microbe interactions that facilitate gut colonization by commensal bifidobacteria
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TY - JOUR - Book Reviews - Ventura, M,Turroni, F,Motherway, MO,MacSharry, J,van Sinderen, D - 2012 - January - Host-microbe interactions that facilitate gut colonization by commensal bifidobacteria - Validated - 1 - () - gut microbiota bifidobacteria host-microbe crosstalk genomics LACTOBACILLUS-RHAMNOSUS GG HUMAN-MILK OLIGOSACCHARIDES EXTENSIVE DNA INVERSIONS TOLL-LIKE RECEPTORS BIFIDUM NCFB 1454 AKKERMANSIA-MUCINIPHILA INTESTINAL MICROBIOTA SYMBIOTIC BACTERIA GENOMIC ANALYSIS IMMUNE-SYSTEM - Microorganisms live in a myriad of ecological niches. The human intestine is among the most densely populated environments; here, a multitude of bacteria appear to have co-evolved to impact beneficially upon the health of their human host. The precise molecular mechanisms and signaling pathways employed by commensal bacteria, including those that facilitate colonization and persistence, remain largely unknown despite the perceived positive effects of such host microbe interactions. In this review we discuss several fascinating relationships between the gastrointestinal tract and commensal bacteria, with particular emphasis on bifidobacteria as a prototypical group of human enteric microorganisms. - 467 - 476 - DOI 10.1016/j.tim.2012.07.002 DA - 2012/01 ER -
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@review{V190495889, = {Book Reviews}, = {Ventura, M and Turroni, F and Motherway, MO and MacSharry, J and van Sinderen, D }, = {2012}, = {January}, = {Host-microbe interactions that facilitate gut colonization by commensal bifidobacteria}, = {Validated}, = {1}, = {()}, = {gut microbiota bifidobacteria host-microbe crosstalk genomics LACTOBACILLUS-RHAMNOSUS GG HUMAN-MILK OLIGOSACCHARIDES EXTENSIVE DNA INVERSIONS TOLL-LIKE RECEPTORS BIFIDUM NCFB 1454 AKKERMANSIA-MUCINIPHILA INTESTINAL MICROBIOTA SYMBIOTIC BACTERIA GENOMIC ANALYSIS IMMUNE-SYSTEM}, = {{Microorganisms live in a myriad of ecological niches. The human intestine is among the most densely populated environments; here, a multitude of bacteria appear to have co-evolved to impact beneficially upon the health of their human host. The precise molecular mechanisms and signaling pathways employed by commensal bacteria, including those that facilitate colonization and persistence, remain largely unknown despite the perceived positive effects of such host microbe interactions. In this review we discuss several fascinating relationships between the gastrointestinal tract and commensal bacteria, with particular emphasis on bifidobacteria as a prototypical group of human enteric microorganisms.}}, pages = {467--476}, = {DOI 10.1016/j.tim.2012.07.002}, source = {IRIS} }
Data as stored in IRIS
OTHER_PUB_TYPE | Book Reviews | ||
AUTHORS | Ventura, M,Turroni, F,Motherway, MO,MacSharry, J,van Sinderen, D | ||
YEAR | 2012 | ||
MONTH | January | ||
TITLE | Host-microbe interactions that facilitate gut colonization by commensal bifidobacteria | ||
RESEARCHER_ROLE | |||
STATUS | Validated | ||
PEER_REVIEW | 1 | ||
TIMES_CITED | () | ||
SEARCH_KEYWORD | gut microbiota bifidobacteria host-microbe crosstalk genomics LACTOBACILLUS-RHAMNOSUS GG HUMAN-MILK OLIGOSACCHARIDES EXTENSIVE DNA INVERSIONS TOLL-LIKE RECEPTORS BIFIDUM NCFB 1454 AKKERMANSIA-MUCINIPHILA INTESTINAL MICROBIOTA SYMBIOTIC BACTERIA GENOMIC ANALYSIS IMMUNE-SYSTEM | ||
REFERENCE | |||
ABSTRACT | Microorganisms live in a myriad of ecological niches. The human intestine is among the most densely populated environments; here, a multitude of bacteria appear to have co-evolved to impact beneficially upon the health of their human host. The precise molecular mechanisms and signaling pathways employed by commensal bacteria, including those that facilitate colonization and persistence, remain largely unknown despite the perceived positive effects of such host microbe interactions. In this review we discuss several fascinating relationships between the gastrointestinal tract and commensal bacteria, with particular emphasis on bifidobacteria as a prototypical group of human enteric microorganisms. | ||
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START_PAGE | 467 | ||
END_PAGE | 476 | ||
DOI_LINK | DOI 10.1016/j.tim.2012.07.002 | ||
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